Pulmonary embolism on cardiac CT

Acute PE on CCTA — central filling defects, saddle emboli, and the right-heart-strain signs that change prognosis. Plus chronic thromboembolic disease.

While standard CT pulmonary angiography (CTPA) is specifically timed to optimize right-sided heart opacification, pulmonary emboli are frequently detected as incidental findings or alternative causes of chest pain on standard Coronary CTAs — which are timed for left-sided enhancement.

1 Acute pulmonary embolism

CT findings. Emboli appear as central filling defects within the opacified pulmonary arteries, maintaining a sharp interface with the contrast material.

A saddle embolus is a massive clot that straddles the bifurcation of the main pulmonary artery.

2 Right heart strain

Massive acute emboli increase pulmonary pressures, resulting in right ventricular (RV) failure. CT signs of right-heart strain — which indicate a poorer prognosis — include:

1
RV-to-LV diameter ratio > 1

The dilated RV becomes the dominant chamber on axial cuts.

2
Leftward bowing or straightening of the interventricular septum

The high RV pressure pushes the septum into the LV, distorting its normal D-shape.

3
Contrast reflux into the IVC and hepatic veins

Hemodynamic backup — contrast spills back through the tricuspid valve into the systemic venous side.

3 Pulmonary infarcts

Peripheral, wedge-shaped opacities — most commonly in the lower lobes — represent ischemic tissue necrosis from the obstructed segmental supply.

4 Chronic Thromboembolic Pulmonary Hypertension (CTEPH)

A small percentage of acute emboli fail to resolve, transforming into organized, fibrotic thromboemboli.

CT findings. Unlike the central defects of acute PE, chronic emboli form eccentric, wall-adherent filling defects that can appear as:

  • Slits
  • Webs
  • Bands

These cause abrupt vessel narrowing rather than the smooth tapering of acute clot.

Secondary changes worth noting:

  • The central main pulmonary artery will be massively dilated (> 2.9 cm) with pruning of the distal peripheral vessels.
  • The lung parenchyma frequently demonstrates a mosaic attenuation pattern — where areas of hypoattenuation correlate with hypoperfusion.
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